Saturday, March 22, 2008

On Correlation versus Causation

Among the common mistakes made by data miners who lack training in statistics is the confusion between correlation and causation, often arising from the difference between observational studies and random controlled trials. To start with, here are some simple definitions.
• There is a relation of (positive) correlation between two random variables when high values of one are likely to be associated with high values of the other.
• There is a relation of cause and effect between two random variables when one is a determinant of the other.
It is easy to see from these definitions that:
• Causation implies correlation, but correlation does not necessarily imply causation.
• Correlation is easy to establish, causation is not.
As a matter of fact:
• Random controlled trials establish causation.
• Observational studies only bring out correlation.
• Simple problems: A solves 81 out of 87 and B solves 234 out of 270
• Complex problems: A solves 192 out of 263 and B solves 55 out of 80
Should this additional information change C's decision as to which company to hire? Of course. Although GoodSoft does better overall, it actually has worse performance on both the simple problems and the complex problems. Such situation are also known as examples of Simpson's paradox. It is a paradox because although mathematically correct, it is somewhat counterintuitive. The "variable" complexity in this example is a confounding variable, because it interacts with the calculated outcome in a way that may easily be overlooked, but may have an adverse effect on conclusions reached. Another well-known instance of Simpson's paradox arises in some of our US presidential elections where the winner does not carry a popular vote (i.e., the tally of individual votes gives the opponent as winner). The confounding variable in this case is the electoral college.
In the medical domain, for example, it is critical to discover causal rather than only correlational relationships. One cannot take the risk of treating the "wrong" cause of a particular ailment. The same is also true of many other situations outside of medicine. Hence, data miners should do well to understand confounding effects and use that knowledge both in the design of the experiments they run and the conclusions they draw from experiments in general. I have been guilty of "jumping the gun" myself and reporting results that clearly ignored possible confounding effects.

Let me turn now to more mundane business applications. In reaction to a (much shorter) comment I posted about the difference between correlation and causation here, a couple of individuals reacted as follows (I reproduce some of that conversation here for completeness):

• (Jaime) - I don't think that insurance companies or any other business that would use data mining would or necessarily should care about the difference between correlation and causation in factors they don't have any control. (exceptions, of course, for anything medical or legal). If they can determine that people with freckles have less car accidents, why shouldn't they offer people with freckles lower rates?
• (Will) - Jamie makes a good point. The question of correlation versus causation will be of only philosophical interest to a data mining practitioner, assuming that the underlying behavior being modeled does not change (and this will often be a safe bet). An illustration should make this subtlety clear. Suppose that insurance data indicates that people who play the board game Monopoly are better life insurance risk than people who do not. An insurance company might very well like to take advantage of such knowledge. Is their necessarily a causal arrow between these two items? No, of course not. Monopoly might not "make" someone live longer, and living longer may not "make" someone play Monpoly. Might there exist another characteristic which gives rise to both of these items (such as being a home-body who avoids death by automobile)? Yes, quite possibly. The insurance company does not care, as long as the relationship continues to hold.

This brings up an interesting point of course. Is the matter of causation versus correlation only a philosophical one, with little bearing in practice (a little bit like the No Free Lunch Theorem is a great theoretical result but seems to have little real impact in practical applications of machine learning; but that is another discussion: here for details on this unrelated but interesting topic). Let me try to address this here a little bit (much of this is also found in my response on the above blog). The statistician (I use the term loosely, I am not a statistician myself) seeks the true cause, the one that remains valid through time. On the other hand, the (business) practitioner seeks mainly utility or applicability, which may become invalid over time but serves him/her right for some reasonable amount of time. Under this view of the world, I think it is possible to reconcile the two perspectives. Indeed, one can see that the statements "assuming that the underlying behavior being modeled does not change" and "as long as the relationship continues to hold" may be interpreted (in some way, see below) as effectively equivalent to what statisticians regard as "controlling for variables". By taking this kind of dynamic approach where the relationship (or behavior) is "continuously" monitored for validity and the action is taken only as long as that relationship holds, the user is, in effect, relieved from the problem of lurking variables. Let me illustrate on Will's example. Statisticians would indeed argue that there may be a confounding variable that explains the insurance company's finding, one that has nothing to do with playing monopoly. Will proposed one: "being a home-body". I'll continue the argument with that one. In this case, it may therefore be that there are more home-body monopoly players than not; and it is the "home-bodyness" (if such a word exists) that explains the lower risk for life insurance (and not the monopoly-playing). Now, a statistician would be right in this case, and if one had to come up with the "correct" answer and build a model that remains accurate for now AND the future, you would have to accept the statistician's approach and build your model using home-bodyness rather than monopoly playing. There is little arguing here. I think that what Will and Jaime are be getting at is that there is a way to, in some sense, side-step this issue; namely: monitor the relationship. Indeed, if I keep on looking and checking that the correlation continues to hold, then I don't care about any confounding effect. If there are none, then the correlation also manifests a causation and I am safe; if there are some confounding effects, they will become manifest over time as the observed correlation is weakened. Hence, I can choose at that time to invalidate my model. But in the meantime, it served me right, was accurate, and I did not worry about controlling anything. Going back to the example, as long as the correlation is strong, I am OK. If it turns out that it is home-bodyness that causes the lower risk, I may eventually see more and more non monopoly players with low risk who also turn out to be home-bodies. In this case the originally observed correlation will decrease telling me that I may wish to discontinue the use of my model.

The distinction may be viewed as only of philosophical interest, at least in the context of such business cases. Again, in medicine, one may have a different perspective as also pointed out by Jaime and Will. One of the drawbacks of the "correlation-driven" approach is that when the model is no longer valid (as seen by the decreasing correlation value), the practitioner has no idea what may be the cause and is thus left with no information as to where to go next. Then again, as suggested by Jaime and Will, maybe he/she does not care. From a strictly business standpoint, he/she was able to quickly build a model with high utility (even if only for a shorter period of time) instead of having to expand a lot of resources to build a "causation" model, with the risk of not doing any better as not all confounding can ever be controlled for! (In fact, there are even situations where the controlled experiments that would be necessary cannot be run; see here for a fun example).

After all is said and done, and more has been said than done :-), one should be aware of confounding effects (or Simpson's paradox), and know how to deal with them: 1) stick to strictly random controlled experiments; or 2) use observations but handle with careful and continuous monitoring.

Christophe Giraud-Carrier said...

I am sorry to comment on my own post, but I thought it would only be fair to mention the fact that Will read my comment on his post and reacted as follows (see the link to Will's blog in my post).
"Will Dwinnell said...
Yes, Christophe, I believe I would agree with everything in your second comment. Thank you!"
It's nice to reach consensus. Thanks Will.

Will Dwinnell said...

I think the whole thing boils down to context. A data mining practitioner may full well acknowledge that the correlation between A and B that he discovers does not necessarily imply that A causes B, but it will not matter as long as the measured correlation is predictive.

Whether A causes B, B causes A or some as-yet unknown factor causes both of them, the practitioner may not care, as long as it holds.

As a practical matter, it is frequently the case that it is much easier for organizations to monitor the quality of their models than it is to hunt for the root cause of observed behaviors.

Yes, I think we agree on these matters. If I am simply repeating what you have written, then I suppose that is a good thing.